Hakima Amri, Ph.D.

Assistant Professor
Department of Physiology and Biophysics
Georgetown University School of Medicine

Co-Investigator,
Educational Initiative in Complementary,
Alternative and Integrative Medicine
3900 Reservoir Road, NW
Washington, D.C. 20007
Phone: 202-687-8594
Fax: 202-687- 7407

E-Mail: amrih@georgetown.edu

Educational and Professional Experience:

University of Constantine, Algeria, B.S.
University Pierre & Marie Curie, Paris, France, M.Sc., Ph.D.
University of Guelph, Ontario, Canada, Research Fellowship
Georgetown University School of Medicine, Postdoctoral Fellowship
Faculty Position held at Georgetown University School of Medicine in Washington, D.C. since 1997

Scholarly Interests:

As a biochemist, cell biologist and reproductive physiologist, my research centers on the biochemistry of steroid hormones, stress regulation, molecular aspects of reproductive endocrinology, and women?s health issues. I am also co-investigator and a founding member of the Complementary, Alternative and Integrative Medicine Initiative at Georgetown University School of Medicine. My efforts are directed at bridging the gap between conventional science and alternative methodologies in order to help physicians of tomorrow understand and better integrate complementary treatment modalities. My commitment to educating research scientists and healthcare providers, as well as the general public make me an advocate for innovative improvement in science and health care.

Personal Background and Interests:

Born and raised in Algeria, my formative years were spent in France and the United States, exposing me to a wide range of scientific and educational philosophies. 

Selected Publications:

• V. Papadopoulos, A. Kapsis, H. Li, H. Amri, M. Hardwick, M. Culty, P. Kasprzyk, M. Carlson, J-P. Moreau, K. Drieu (2000). Drug-induced inhibition of the peripheral-type benzodiazepine receptor expression and cell proliferation in human breast cancer cells. Anticancer Res. 20: 2853-2848.
  
• H. Amri, K. Drieu, V. Papadopoulos (2003) Transcriptional suppression of adrenal cortical peripheral-type benzodiazepine receptor gene and inhibition of steroid synthesis by ginkgolide B. Biochemical Pharmacology 65: 717-729.
  
•  V. Papadopoulos, H. Amri, H. Li, Z. Yao, R.C. Brown, B. Vidic, M. Culty (2001) Structure, function and regulation of the mitochondrial peripheral-type benzodiazepine receptor. Thérapie, 56:549-556.
  
• Amri H, Drieu K and Papadopoulos V (2002) Use of ginkgolide B and a ginkgolide-activated response element to control gene transcription: example of the adrenocortical peripheral-type benzodiazepine receptor. Cellular and Molecular Biology, 48: 633-639.

For more publications, go to PubMed at the following link:

http://www.ncbi.nlm.nih.gov/entrez/